Uchigata Y, Hirata Y, Omori Y (1995) A novel concept of type VII hypersensitivity introduced by insulin autoimmune syndrome (Hirata’s disease). (2014) Insulin autoimmune syndrome (Hirata Disease) in European Caucasians taking Hirata Y, Ishizu H, Ouchi N et al. Horm Res …
Dozio N, Scavini M, Beretta A et al. (1992) Strong association of insulin autoimmune syndrome with HLA-DR4. (2013) A novel atypical presentation of insulin autoimmune syndrome (Hirata’s disease) in a child. Introduction. Balestrieri A, Magnani E, Ragazzini C, Pasini G (2015) Primary insulin autoimmune syndrome in an Italian woman: a case report.
VKH may variably also involve the inner ear, with effects on hearing, the skin and the meninges of the central nervous system. di Endocrinologia, Università degli Studi di Padova, Via Ospedale Civile 105, 35128, Padova, ItaliaU.O.C. Intern Med 52:369–372 (2013) A case of autoimmune hypoglycemia outside Japan: rare, but in the era of expanding drug-list, important to suspect. Lupsa BC, Chong AY, Cochran EK et al. Gopal K, Priya G, Gupta N et al. Diabetes Res Clin Pract 22:89–94 Betterle, C., Presotto, F. La sindrome di Hirata (stato dell’arte). 2002 Apr. J Jpn Diabetes Soc 13:312–320 Intern Med.
(2005) CT60 single nucleotide polymorphisms of the cytotoxic T-lymphocyte-associated antigen-4 gene region is associated with Graves’ disease in an Italian population. Insulin autoimmune syndrome (IAS, Hirata disease) is an unusual cause of hypoglycemia.
Although IAS is considered to be the third greatest cause of hypoglycemia in Japan, only a handful of cases … Tale sindrome è caratterizzata da un’ipoglicemia autoimmune dovuta alla presenza di autoanticorpi anti-insulina che si formano spontaneamente o in seguito al contatto dei soggetti con farmaci con un gruppo sulfidrilico o all’acido alfa-lipoico. Cugno M, Castelli R, Marco Cicardi M (2008) Angioedema due to acquired C1-inhibitor deficiency: a bridging condition between autoimmunity and lymphoproliferation. (1992) An unusual case of recurrent hypoglycaemia: 10-year follow up of a child with insulin auto-immunity. (2007) Possible relevance of alpha lipoic acid contained in a health supplement in a case of insulin autoimmune syndrome. Nella storia naturale delle malattie autoimmuni sono implicati diversi meccanismi autoanticorpo-mediati e/o cellulo-mediati. Dove Medical Press
Gullo D, Evans JL, Sortino G et al. Ital J Med 9:169–172 (1993) Patients with Graves’ disease who developed insulin autoimmune syndrome (Hirata disease) possess HLA-Bw62/Cw4/DR4 carrying DRB1
Available therapies may include drugs that reduce pancreatic insulin secretion (somatostatin analogues and diazoxide, for instance) and immunosuppressive agents (glucocorticoids, azathioprine and rituximab). Blackwell Sci, Oxford, pp 761–781 Thyroid 15:232–238 Diabetes Res Clin Pract 83:e19–e20 Uchigata Y, Kuwata S, Tsushima T et al.
You can also search for this author in Please enable it to take advantage of the complete set of features! Autoimmunity Reviews 8:156–159 Insulin autoimmune syndrome in a health supplement user: the effectiveness of cornstarch therapy for treating hypoglycemia. Coombs RR, Gell PG (1975) Classification of allergic reactions responsible for clinical hypersensitivity and disease. Savas-Erdeve S, Yılmaz Agladioglu S, Onder A, et al. The diagnosis of IAS is challenging, requiring a careful workup aimed at excluding other causes of hyperinsulinemic hypoglycemia.
(2007) Insulin autoimmune syndrome possibly caused by alpha lipoic acid. Tra questi vi è il meccanismo di tipo VII caratterizzato dalla presenza di un anticorpo che lega un ormone. Uchigata Y (2007) The novel agent, alpha-lipoic acid, can cause the development if insulin autoimmune syndrome. The estimated prevalence was 0.017 per 100,000 population. Kiyotaka Hirata et al. Epub 2016 Nov 17. Uchigata Y, Tokunaga K, Nepom G et al. 2018 Sep;6(17):335. doi: 10.21037/atm.2018.07.32.Cappellani D, Sardella C, Campopiano MC, Falorni A, Marchetti P, Macchia E.Endocrinol Diabetes Metab Case Rep. 2018;2018:18-0122. doi: 10.1530/EDM-18-0122. Indian J Endocrinol Metab 17:1117–1119 The median age of onset was 63 years (range 3–88 years; Figure 4). J Clin Endocrinol Metab 83:643–648 Unable to load your delegates due to an error
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